Preliminary studies suggest conolidine may well inhibit particular ion channels, minimizing neuronal excitability and limiting soreness alerts. This mechanism is particularly relevant in neuropathic suffering, wherever irregular signaling leads to persistent discomfort. Additionally, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to discomfort percept... https://davidw159pgx3.webbuzzfeed.com/profile